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- W1652803474 abstract "^ Chronic Hepatocanalicular Intrahepatic Cholestasis due to Verpamil (A Case Report)Cholestatic liver diseases arise as disorders of bile flow resulting from diverse etiologies. Many drugs have been reported to be associated with cholestasis. The clinical and pathologic presentation of cholestasis has been divided into canalicular and hepatocanalicular or exudative cholestasis. Many drugs produce inflammatory cholestasis either lobular or portal, including chlorpromazine, eiythromycin, captopril, trimethoprim-sulfamethoxazole, amoxicillin/clavulanic acid, imipramine, methimazole, the oral sulfonylurea and many others. The mechanism involved generally is unknown. Hepatotoxicity due to verapamil is very rare and to the best of our knowledge only 11 cases have been reported. We report a 67 year old man developed cholestatic liver injury with verapamil (120 mg/day) for coronary artery disease Kolestatlk karaciger hastaligi cesitli etyolojilerin sonucu olusan safra akimi bo-zukluklariyla ortaya cikar. Bircok ilacin kolestaz ile iliskili oldugu bildirilmistir. Klinik ve patolojik gorunumune gore kolestaz; kanalikuler ve hepatokanalikuler veya ek-sudatif' kolestaz olarak siniflandirilir. Bircok ilac ya portal ya da lobuler infiamatuvar ko-lestaza yol acarlar, bu ilaclar; klorpromazin, eritromisin, kaptopril, trimetoprim-sulfametaksazol, amoksisilin/klavulanik asit, imipramin, metimazol. oral sulfanureler ve diger bir cok ilaci kapsar. Sorumlu mekanizma siklikla bilinmez. Verapamil'e bagli he-patotoksisite cok nadirdir ve en yeni bilgilerimize gore sadece 11 vaka bildirilmistir . Biz 67 yasinda, koroner arter hastaligi nedeniyle verapamil (120 mg/gun) kullanan bir erkek hastada kolestatik karaciger hastaligi gelistigini rapor ediyoruz." @default.
- W1652803474 created "2016-06-24" @default.
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- W1652803474 date "2001-01-01" @default.
- W1652803474 modified "2023-09-22" @default.
- W1652803474 title "Verapamil’e Bağlı Kronik Hepatokanaliküler Tip Intrahepatik Kolestaz Olgusu" @default.
- W1652803474 doi "https://doi.org/10.5835/jecm.v18i2.343" @default.
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