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- W1664488112 startingPage "837" @default.
- W1664488112 abstract "Cardiovascular abnormalities accompany both portal hypertension and cirrhosis. These consist of hyperdynamic circulation, defined as reduced mean arterial pressure and systemic vascular resistance, and increased cardiac output. Despite the baseline increased cardiac output, ventricular inotropic and chronotropic responses to stimuli are blunted, a condition known as cirrhotic cardiomyopathy. Both conditions may play an initiating or aggravating pathogenic role in many of the complications of liver failure or portal hypertension including ascites, variceal bleeding, hepatorenal syndrome and increased postoperative mortality after major surgery or liver transplantation. This review briefly examines the major mechanisms that may underlie these cardiovascular abnormalities, concentrating on nitric oxide, endogenous cannabinoids, central neural activation and adrenergic receptor changes. Future work should address the complex interrelationships between these systems." @default.
- W1664488112 created "2016-06-24" @default.
- W1664488112 creator A5015064556 @default.
- W1664488112 creator A5042678528 @default.
- W1664488112 creator A5067383696 @default.
- W1664488112 date "2006-01-01" @default.
- W1664488112 modified "2023-10-03" @default.
- W1664488112 title "Cardiac and vascular changes in cirrhosis: Pathogenic mechanisms" @default.
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- W1664488112 doi "https://doi.org/10.3748/wjg.v12.i6.837" @default.
- W1664488112 hasPubMedCentralId "https://www.ncbi.nlm.nih.gov/pmc/articles/4066146" @default.
- W1664488112 hasPubMedId "https://pubmed.ncbi.nlm.nih.gov/16521209" @default.
- W1664488112 hasPublicationYear "2006" @default.
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