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- W166893722 abstract "Inflammatory diseases place a heavy burden on the American health care system. Tristetraprolin, a zinc-dependent mRNA binding protein decreases the stability of mRNAS coding for some proinflammatory cytokines. Tristetraprolin-deficient mice develop a profound inflammatory syndrome. Tristetraprolin is a potential cancer therapy due to its control of vascular endothelial growth factor mRNA stability. Cinnamon extract stimulates the expression of antiinflammatory tristetraprolin. Bioactive compound(s) in cinnamon extract define its molecular mechanisms. Cinnamon is potentially important in tristetraprolin-mediated inflammatory diseases." @default.
- W166893722 created "2016-06-24" @default.
- W166893722 creator A5065528836 @default.
- W166893722 date "2010-01-01" @default.
- W166893722 modified "2023-09-26" @default.
- W166893722 title "Cinnamon and Immune Actions: Potential Role in Tristetraprolin-Mediated Inflammatory Diseases" @default.
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- W166893722 doi "https://doi.org/10.1007/978-1-60761-061-8_30" @default.
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