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- W1683237191 abstract "Lytic infections induce direct cell or tissue destruction leading to such diseases as hepatitis, encephalitis, diabetes, among many others. Polioencephalomyelitis triggered by infection with lactate dehydrogenase–elevating virus (LDV) in immunosuppressed mice that are co-infected with a retrovirus is a direct consequence of lytic infection of motor neurons in the spinal cord. Similar pathologies may also follow indirect tissue destruction, mediated by immune mechanisms initially directed against the invading virus. For example, the lethal neurological disease induced by lymphocytic choriomeningitis virus (LCMV) is a consequence of killing of virallyinfected cells by LCMV-specific cytolytic T lymphocytes (reviewed in Ref. 2). Many virally-induced autoimmune diseases may also result from either cross-reactivity between viral and self-epitopes or spreading of an antiviral response to self-antigens. Finally, pathologies may be caused by nonspecific bystander effects of infections, like cytokine secretion. Here, we report how LDV may exacerbate autoantibody-mediated diseases through such a mechanism. Special emphasis is put on the role of gammainterferon (IFN-γ) production on the outcome of concomitant autoantibody-mediated autoimmune diseases such as hemolytic anemia and thrombocytopenic purpura." @default.
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- W1683237191 date "2006-01-01" @default.
- W1683237191 modified "2023-09-27" @default.
- W1683237191 title "Gamma-Interferon Involvement in the Pathogenesis of Lactate Dehydrogenase–Elevating Virus Infection" @default.
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- W1683237191 doi "https://doi.org/10.1007/978-0-387-33012-9_79" @default.
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