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- W169113375 abstract "Publisher Summary This chapter describes the endocrine problems in pregnancy. The thyroid gland enlarges during pregnancy as a result of increased vascularity of the gland, which is associated with a 20% increase in the basal metabolic rate. The enlargement of the thyroid in normal pregnancy is also due to hyperplasia of the glandular tissue. This hyperplasia occurs for four separate reasons. First, the increased renal loss of iodide coupled with the fetal competition for iodide results in a mild iodine-deficiency state. This state sensitizes the gland to the growth-promoting properties of thyroid stimulating hormone. Second, there is a necessity for an increased secretion of thyroid hormone to saturate the increased quantity of thyroid-binding globulin. Third, there is an increase in plasma thyrotropin-releasing hormone level in normal pregnancy and these results in thyroid hyperplasia. Fourth, thyroid stimulation occurs because of an increased level of human chorionic gonadotrophin in plasma. In the first trimester of pregnancy, hypothyroidism results in a high incidence of abortion. It is found that when iodine deficiency exists during the first trimester of pregnancy and the mother is euthyroid, the baby is often born with mental handicap, deafness, and a form of spasticity but often does not have a goiter." @default.
- W169113375 created "2016-06-24" @default.
- W169113375 creator A5062646445 @default.
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- W169113375 date "1993-01-01" @default.
- W169113375 modified "2023-09-27" @default.
- W169113375 title "Endocrine Problems in Pregnancy" @default.
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- W169113375 doi "https://doi.org/10.1016/b978-0-7506-1390-3.50013-1" @default.
- W169113375 hasPublicationYear "1993" @default.
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