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- W169610125 abstract "Obesity is a serious medical problem worldwide and disruption of metabolic/energy homeostasis plays a pivotal role in this global epidemic. In obese people, fatty liver (steatosis) develops, which increases the risk for diabetes, cardiovascular disease, and even, liver cancer. Sirtuin 1 (SIRT1) is a NAD+-dependent deacetylase that functions as a key metabolic/energy sensor and mediates homeostatic responses to nutrient availability. Accumulating evidence indicates that SIRT1 is a master regulator of the transcriptional networks that control hepatic lipid metabolism. During energy-deprived conditions, SIRT1 deacetylates and alters the expression and activities of key transcriptional regulators involved in hepatic lipogenesis, fatty acid β-oxidation, and cholesterol/bile acid metabolism. This review will discuss the latest advances in this field, focusing on beneficial roles of SIRT1 in hepatic lipid metabolism including its potential as a therapeutic target for treatment of steatosis and other obesity-related metabolic diseases." @default.
- W169610125 created "2016-06-24" @default.
- W169610125 creator A5008172153 @default.
- W169610125 creator A5011242470 @default.
- W169610125 creator A5059760054 @default.
- W169610125 date "2013-01-01" @default.
- W169610125 modified "2023-10-18" @default.
- W169610125 title "Sirtuin 1 Deacetylase" @default.
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- W169610125 doi "https://doi.org/10.1016/b978-0-12-407766-9.00016-x" @default.
- W169610125 hasPubMedCentralId "https://www.ncbi.nlm.nih.gov/pmc/articles/4976444" @default.
- W169610125 hasPubMedId "https://pubmed.ncbi.nlm.nih.gov/23374725" @default.
- W169610125 hasPublicationYear "2013" @default.
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