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- W1701460655 abstract "An expanding spectrum of acute and chronic inflammatory diseases is considered 'autoinflammatory' diseases. This review considers autoinflammatory diseases as being distinct from 'autoimmune' diseases. Autoimmune diseases are associated with dysfunctional T cells and treated with 'biologicals', including antitumour necrosis factorα, CTLA-Ig, anti-IL-12/23, anti-CD20, anti-IL-17 and anti-IL-6 receptor. In contrast, autoinflammatory diseases are uniquely attributed to a dysfunctional monocyte caspase 1 activity and secretion of IL-1β; indeed, blocking IL-1β results in a rapid and sustained reduction in the severity of most autoinflammatory diseases. Flares of gout, type 2 diabetes, heart failure and smouldering multiple myeloma are examples of seemingly unrelated diseases, which are uniquely responsive to IL 1β neutralization." @default.
- W1701460655 created "2016-06-24" @default.
- W1701460655 creator A5028234430 @default.
- W1701460655 date "2010-12-16" @default.
- W1701460655 modified "2023-09-27" @default.
- W1701460655 title "Blocking interleukin-1β in acute and chronic autoinflammatory diseases" @default.
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- W1701460655 doi "https://doi.org/10.1111/j.1365-2796.2010.02313.x" @default.
- W1701460655 hasPubMedCentralId "https://www.ncbi.nlm.nih.gov/pmc/articles/3074355" @default.
- W1701460655 hasPubMedId "https://pubmed.ncbi.nlm.nih.gov/21158974" @default.
- W1701460655 hasPublicationYear "2010" @default.
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