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- W17191877 abstract "The peroxisome proliferator-activated receptors (PPARs) are members of the nuclear receptor superfamily whose activity can be modulated by polyunsaturated fatty acids, prostaglandins, eicosanoids, and various synthetic ligands []. There are three PPARs, termed α, δ and λ []. PPARα has been shown to regulate the Β- and ω-oxidation of fatty acids in the liver and heart, and fibric acids, a class of hypolipidemic drugs, are synthetic ligands for PPARα []. PPARδ, which is expressed in a wide variety of tissues, has been shown to be important in embryo implantation, cellular proliferation, and HDL metabolism [,]. Although the PPARγ subtype was initially identified in adipocytes [,] and shown to be a key regulator of adipocyte differentiation and glucose homeostasis [, , , ], it is also expressed in other tissues, including placenta, breast, colon, and hematopoetic cells []. A variety of ligands can serve as activators for PPARγ, including the widely prescribed antidiabetic drugs, the TZDs, and metabolites of the cyclooxygenase pathway, such as 15-deoxyΔ12−14 prostaglandin J2 (15d-PGJ2) [, , ]." @default.
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- W17191877 date "2002-01-01" @default.
- W17191877 modified "2023-10-18" @default.
- W17191877 title "Functions of PPAR Gamma in Macrophages and Atherosclerosis" @default.
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- W17191877 doi "https://doi.org/10.1007/978-1-4615-1171-7_3" @default.
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