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- W172000330 abstract "Vitamin D is a precursor molecule that can be obtained from the diet or synthesized in the skin by a process that requires ultraviolet radiation from sunlight. Vitamin D is subsequently activated within the body by two hydroxylations in the liver and kidney to yield the hormone 1,25-dihydroxyvitamin D or calcitriol. Calcitriol acts via the classical steroid hormone pathway through nuclear receptors to regulate the level of target gene expression. Calcitriol has many actions in the body but most prominently it controls calcium and phosphate absorption in the intestine. This action determines the level of circulating mineral ions and their delivery to bone-forming sites to mineralize bone. If vitamin D is deficient or its synthesis or action is defective, adequate mineral is not delivered to bone-forming sites and thus abnormal undermineralized bone is formed. This leads to the metabolic bone disease of rickets in childhood or osteomalacia in adults." @default.
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- W172000330 date "2004-01-01" @default.
- W172000330 modified "2023-10-14" @default.
- W172000330 title "Vitamin D Deficiency, Rickets, and Osteomalacia" @default.
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- W172000330 doi "https://doi.org/10.1016/b0-12-475570-4/01383-4" @default.
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