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- W1723551566 abstract "Vascular inflammation, especially at the level of endothelial cells, has been shown to play a pivotal role in the inception, progression, and clinical complications of atherosclerosis. The common denominators for the activation of inflammatory genes appear to be a small subset of transcription factors—among which include nuclear factor‐κB, activator protein‐1 (AP‐1), and GATA—that function as the central hub of vascular inflammation. Strategies directed to inhibit both the secondary mediators and the primary triggers (atherosclerosis risk factors) appear viable to inhibit atherosclerosis. However, attempts have now been made to address the central hub of vascular inflammation. “Old” drugs, such as dipyridamole, can also now be revisited for properties related to inhibition of vascular inflammation, probably by acting on the common hub of inflammation." @default.
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- W1723551566 date "2010-10-01" @default.
- W1723551566 modified "2023-09-28" @default.
- W1723551566 title "Pharmacological modulation of vascular inflammation in atherothrombosis" @default.
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- W1723551566 doi "https://doi.org/10.1111/j.1749-6632.2010.05784.x" @default.
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