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• W1736165452 abstract "The persistence of memory lymphocytes is a critical feature of adaptive immunity. The TNF family ligand 4–1 BBL supports the antigen‐independent survival of CD 8+ memory T cells. Here, we show that mice lacking 4–1 BB only on αβ T cells show a similar defect in CD 8+ T ‐cell recall responses, as previously shown in 4–1 BBL ‐deficient mice. We show that 4–1 BB is selectively expressed on BM CD 8+ but not CD 4 + memory T cells of unimmunized mice. Its ligand, 4–1 BBL , is found on VCAM ‐1+ stromal cells, CD 11c+ cells, and a G r1 lo myeloid population in unimmunized mice. Adoptive transfer of in vitro generated memory T cells into mice lacking 4–1 BBL only on radioresistant cells recapitulates the defect in CD 8 + T ‐cell survival seen in the complete knockout mice, with smaller effects of 4–1 BBL on hematopoietic cells. In BM, adoptively transferred D s R ed CD 8+ memory T cells are most often found in proximity to VCAM ‐1+ cells or G r1+ cells, followed by B 220+ cells and to a much lesser extent near CD 11c+ cells. Thus, a VCAM ‐1+ CD 45 − stromal cell is a plausible candidate for the radioresistant cell that provides 4–1 BBL to CD 8+ memory T cells in the BM ." @default.
• W1736165452 created "2016-06-24" @default.
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• W1736165452 date "2012-09-20" @default.
• W1736165452 modified "2023-10-16" @default.
• W1736165452 title "Contribution of 4-1BBL on radioresistant cells in providing survival signals through 4-1BB expressed on CD8+ memory T cells in the bone marrow" @default.
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• W1736165452 doi "https://doi.org/10.1002/eji.201242503" @default.
• W1736165452 hasPubMedId "https://pubmed.ncbi.nlm.nih.gov/22886791" @default.
• W1736165452 hasPublicationYear "2012" @default.
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