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- W173678377 endingPage "24" @default.
- W173678377 startingPage "19" @default.
- W173678377 abstract "In acute pancreatitis, initiating cellular events causing acinar cell injury includes co-localization of zymogens with lysosomal hydrolases, leading to premature enzyme activation and pathological exocytosis of zymogens into the interstitial space. This is followed by processes that accentuate cell injury; triggering acute inflammatory mediators, intensifying oxidative stress, compromising the microcirculation and activating a neurogenic feedback. Such localized events then progress to a systemic inflammatory response leading to multiorgan dysfunction syndrome with resulting high morbidity and mortality. The present review discusses some of the most recent insights into each of these cellular processes postulated to cause or propagate the process of acute pancreatitis, and also the role of alcohol and genetics." @default.
- W173678377 created "2016-06-24" @default.
- W173678377 creator A5038899538 @default.
- W173678377 creator A5060106697 @default.
- W173678377 date "2007-01-01" @default.
- W173678377 modified "2023-10-16" @default.
- W173678377 title "Recent insights into the cellular mechanisms of acute pancreatitis" @default.
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