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• W1737157464 abstract "An increase in cytosolic ATP following glucose metabolism by pancreatic beta-cells is the key signal initiating insulin secretion by causing blockade of ATP-dependent K+ channels (KATP). This induces membrane depolarization, leading to an elevation in cytosolic Ca2+ ([Ca2+]i) and insulin secretion. In this report we identify the critical metabolic step by which glucose initiates changes in beta-cell KATP channel activity, membrane potential, and [Ca2+]i. The signal stems from the glycolytic production of NADH during the oxidation of glyceraldehyde 3-phosphate, which is subsequently processed into ATP by mitochondria via the operation of discrete shuttle systems." @default.
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• W1737157464 date "1994-04-01" @default.
• W1737157464 modified "2023-10-16" @default.
• W1737157464 title "Dependence on NADH produced during glycolysis for beta-cell glucose signaling" @default.
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• W1737157464 doi "https://doi.org/10.1016/s0021-9258(19)78078-8" @default.
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