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- W17383219 abstract "Salmonella serotypes can colonize and infect an enormous range of vertebrate hosts after orally ingestion. Infection results in a wide spectrum of acute and chronic illnesses; including gastroenteritis, enteric (typhoid) fever, and bacteremia.1 The consequence of Salmonellae ingestion by vertebrates depends on the serotype. Certain Salmonella serotypes have very narrow host specificity while others infect a wide variety of animal hosts.1 Examples of host-specific serotypes include S. typhi (man), S. pullorum (fowl), and S. arizonae (reptiles). Such serotypes, with rare exceptions, are only pathogenic for a specific host. In contrast S. typhimurium infects a wide variety of hosts, though disease manifestation varies. In immunocompetent humans an acute self limited gastroenteritis is seen after S. typhimurium ingestion, while in mice a systemic illness similar to human enteric fever occurs. In addition host immune status dramatically alters disease outcome. Humans with the acquired immunodeficiency syndrome (AIDS) are highly susceptible to severe systemic infection with S. typhimurium2 and the susceptibility of inbred mice to this serotype varies widely as a result of mutations that alter immune function.3,4" @default.
- W17383219 created "2016-06-24" @default.
- W17383219 creator A5079322424 @default.
- W17383219 date "1995-01-01" @default.
- W17383219 modified "2023-09-23" @default.
- W17383219 title "PhoP/PhoQ: Regulating Salmonella Adaptation to Host Microenvironments" @default.
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- W17383219 doi "https://doi.org/10.1007/978-3-662-22406-9_5" @default.
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