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• W1747957777 abstract "Respiratory syncytial virus (RSV) infection of airway epithelial cells results in persistent NF-kappaB activation and NF-kappaB-mediated interleukin-8 production. Previous studies in airway epithelial cells demonstrated that tumor necrosis factor alpha (TNF-alpha)-induced NF-kappaB activation is transient due to regulation by IkappaBalpha. However, during RSV infection, IkappaBalpha has only a partial inhibitory effect on NF-kappaB activation. Studies presented here demonstrate that neither increased IkappaBalpha production which occurs as a result of RSV-induced NF-kappaB activation nor inhibition of proteasome-mediated IkappaBalpha degradation results in a reversal of RSV-induced NF-kappaB activation. Thus, while manipulation of IkappaBalpha results in reversal of TNF-alpha-induced NF-kappaB activation, manipulation of IkappaBalpha does not result in a reversal of RSV-induced NF-kappaB activation." @default.
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• W1747957777 date "1999-05-01" @default.
• W1747957777 modified "2023-10-14" @default.
• W1747957777 title "Incomplete Regulation of NF-κB by IκBα during Respiratory Syncytial Virus Infection in A549 Cells" @default.
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• W1747957777 doi "https://doi.org/10.1128/jvi.73.5.4502-4507.1999" @default.
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