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- W1789772972 endingPage "215" @default.
- W1789772972 startingPage "205" @default.
- W1789772972 abstract "Summary: Multiple sclerosis (MS) is a disease of the central nervous system (CNS) characterized by inflammatory, demyelinating lesions localized in the brain and spinal cord. Experimental autoimmune encephalomyelitis (EAE) is an animal model of MS that is induced by activating myelin-specific T cells and exhibits immune cell infiltrates in the CNS similar to those seen in MS. Both MS and EAE exhibit disease heterogeneity, reflecting variations in clinical course and localization of lesions within the CNS. Collectively, the differences seen in MS and EAE suggest that the brain and spinal cord function as unique microenvironments that respond differently to infiltrating immune cells. This review addresses the roles of the cytokines interferon-γ and interleukin-17 in determining the localization of inflammation to the brain or spinal cord in EAE." @default.
- W1789772972 created "2016-06-24" @default.
- W1789772972 creator A5035095164 @default.
- W1789772972 creator A5040473930 @default.
- W1789772972 creator A5065844156 @default.
- W1789772972 creator A5074888016 @default.
- W1789772972 date "2012-06-21" @default.
- W1789772972 modified "2023-09-27" @default.
- W1789772972 title "Mechanisms regulating regional localization of inflammation during CNS autoimmunity" @default.
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