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- W1796126005 abstract "Classical genetic and molecular data show that genes determining disease resistance in plants are frequently clustered in the genome. Genes for resistance ( R genes) to diverse pathogens cloned from several species encode proteins that have motifs in common. These motifs indicate that R genes are part of signal-transduction systems. Most of these R genes encode a leucine-rich repeat (LRR) region. Sequences encoding putative solvent-exposed residues in this region are hypervariable and have elevated ratios of nonsynonymous to synonymous substitutions; this suggests that they have evolved to detect variation in pathogen-derived ligands. Generation of new resistance specificities previously had been thought to involve frequent unequal crossing-over and gene conversions. However, comparisons between resistance haplotypes reveal that orthologs are more similar than paralogs implying a low rate of sequence homogenization from unequal crossing-over and gene conversion. We propose a new model adapted and expanded from one proposed for the evolution of vertebrate major histocompatibility complex and immunoglobulin gene families. Our model emphasizes divergent selection acting on arrays of solvent-exposed residues in the LRR resulting in evolution of individual R genes within a haplotype. Intergenic unequal crossing-over and gene conversions are important but are not the primary mechanisms generating variation." @default.
- W1796126005 created "2016-06-24" @default.
- W1796126005 creator A5017632047 @default.
- W1796126005 creator A5021990364 @default.
- W1796126005 date "1998-11-01" @default.
- W1796126005 modified "2023-10-18" @default.
- W1796126005 title "Clusters of Resistance Genes in Plants Evolve by Divergent Selection and a Birth-and-Death Process" @default.
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