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- W179929576 endingPage "184" @default.
- W179929576 startingPage "153" @default.
- W179929576 abstract "Free radicals are not biological curiosities or anomalies but are byproducts of cellular physiology generated by specific enzymes, by autoxidation, and by energy transfer reactions in all life forms using oxygen. Under physiological conditions, sophisticated antioxidant defense systems prevent the reactive O2 metabolites from potential injurious interactions with cell components critical for the viability of cells. However, under the conditions of hypoxia/ischemia or reoxygenation/reperfusion, the equilibrium between the O2 radical and its antioxidant defense system is lost. This imbalance leads to radical-induced cell damage (1). This chapter contains the mechanism of toxicity posed by oxygen stresses and the responses of brain cells to these common etiologies that may happen in stroke." @default.
- W179929576 created "2016-06-24" @default.
- W179929576 creator A5054251357 @default.
- W179929576 date "1997-01-01" @default.
- W179929576 modified "2023-09-25" @default.
- W179929576 title "Free Radical-Mediated Cerebral Damage After Hypoxia/Ischemia and Stroke" @default.
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