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- W1799631694 abstract "Abstract Using IgG immune complex deposition to trigger acute lung inflammation in rats, we have previously shown that exogenously administered IL-13 suppresses the acute inflammatory response. In the same model, expression of both mRNA and protein for IL-13 has now been detected. Treatment of rats with Ab to IL-13 accentuated the inflammatory response, with significant increases in lung vascular permeability and in the number of neutrophils in bronchoalveolar lavage fluids. In the presence of anti-IL-13, activation of the transcription factor, NF-κB, was significantly increased in lung. In addition, anti-IL-13 caused significant increases in bronchoalveolar lavage levels of TNF-α, macrophage inflammatory protein-2, and cytokine-inducible neutrophil chemoattractant but no changes in lung vascular ICAM-1. These data suggest that during lung inflammation endogenous IL-13 regulates NF-κB activation and related cytokine/chemokine generation, all of which determines the intensity of the lung inflammatory response." @default.
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- W1799631694 date "1999-01-15" @default.
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- W1799631694 title "Regulation of Acute Lung Inflammatory Injury by Endogenous IL-13" @default.
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- W1799631694 doi "https://doi.org/10.4049/jimmunol.162.2.1071" @default.
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