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- W1803433130 abstract "<h2>Abstract</h2> <i>Background</i>. Leukocytes are associated with myocardial injury during reperfusion after ischemia. Short periods of leukocyte depletion during reperfusion result in persistent attenuation of postischemic myocardial dysfunction. <i>Methods</i>. Leukocyte depletion was examined in a canine model of regional myocardial ischemia and reperfusion. The extracorporeal circuit and cardioplegia circuits underwent leukocyte depletion by mechanical filtration. Animals were instrumented for baseline global function before 90-minute occlusion of the left anterior descending coronary artery. Global function during ischemia and at 5, 30, 60, and 90 minutes after a 60-minute cardioplegic arrest using continuous blood cardioplegia was assessed in leukocyte-depleted (n = 9) and control (n = 10) groups. <i>Results</i>. No significant difference between groups was seen for systemic leukocyte counts, global function, or water content. Endothelial function was significantly protected as assessed by response to both calcium ionophore (endothelial-dependent, receptor-independent relaxation: leukocyte-depleted, 72% ± 19% of endothelin-induced constriction versus control, 46% ± 14%; <i>p</i> < 0.05) and acetylcholine (endothelial-dependent, receptor-dependent relaxation: leukocyte-depleted, 83% ± 11% versus control, 44% ± 15%; <i>p</i> < 0.05). <i>Conclusions</i>. Leukocyte-mediated endothelial reperfusion injury can be attenuated by leukocyte depletion during reperfusion." @default.
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- W1803433130 title "Leukocyte Depletion of Blood Cardioplegia Attenuates Reperfusion Injury11Presented at the Thirty-Second Annual Meeting of The Society of Thoracic Surgeons, Orlando, FL, Jan 29–31, 1996." @default.
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- W1803433130 doi "https://doi.org/10.1016/s0003-4975(96)00736-9" @default.
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