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• W180428094 endingPage "340" @default.
• W180428094 startingPage "313" @default.
• W180428094 abstract "Carcinogenesis results from the long-term accumulation of diverse structural and functional molecular alterations. These changes are sequentially expressed within each, and ultimately across all, levels of biologic organization (e.g., from DNA and RNA, to protein, organelle, cell, tissue, and organ levels) (1). This multistep process typically involves an array of known molecular targets (e.g., APC, k-ras, c-myc, MLH1/MSH2/MSH6, PTEN, p53, EGFR, IGF-1, COX, β-catenin), and undoubtedly others that are not yet identified. Nevertheless, current epidemiologic and experimental evidence suggests that many tumors are associated with the overexpression of COX-2, the inducible COX isozyme that catalyzes a crucial step in prostaglandin synthesis. Indeed, COX-2 is overexpressed in stromal, endothelial, or epithelial tumor cells and appears to be important in all stages of tumori-genesis; hence it may be targeted for either cancer prevention or therapy." @default.
• W180428094 created "2016-06-24" @default.
• W180428094 creator A5008765045 @default.
• W180428094 creator A5015008352 @default.
• W180428094 creator A5032280731 @default.
• W180428094 creator A5080636708 @default.
• W180428094 date "2003-01-01" @default.
• W180428094 modified "2023-09-23" @default.
• W180428094 title "Potential Role of NSAIDs and COX-2 Blockade in Cancer Therapy" @default.
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