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• W1805625419 startingPage "5157" @default.
• W1805625419 abstract "Abstract Largely viewed as proinflammatory, innate responses combine with adaptive immunity to generate the most effective form of antifungal resistance, and T cells exercise feedback control over diverse effects of inflammation on infection. Some degree of inflammation is required for protection, particularly in mucosal tissues, during the transitional response occurring between the rapid innate and slower adaptive response. However, progressive inflammation worsens disease and ultimately prevents pathogen eradication. IDO, tryptophan catabolites (“kynurenines”), and regulatory T cells help to tame overzealous and exaggerated inflammatory responses. In this context, IL-23 and the Th17 pathway, which down-regulate tryptophan catabolism, may instead favor pathology and serve to accommodate the seemingly paradoxical association of chronic inflammation with fungal persistence. Recent data support a view in which IL-23/IL-17 antagonistic strategies, including the administration of synthetic kynurenines, could represent a new means of harnessing progressive or potentially harmful inflammation." @default.
• W1805625419 created "2016-06-24" @default.
• W1805625419 creator A5036706420 @default.
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• W1805625419 creator A5079557352 @default.
• W1805625419 creator A5087834518 @default.
• W1805625419 date "2008-04-15" @default.
• W1805625419 modified "2023-10-05" @default.
• W1805625419 title "IL-17 and Therapeutic Kynurenines in Pathogenic Inflammation to Fungi" @default.
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• W1805625419 doi "https://doi.org/10.4049/jimmunol.180.8.5157" @default.
• W1805625419 hasPubMedId "https://pubmed.ncbi.nlm.nih.gov/18390695" @default.
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