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• W1808449080 endingPage "2427" @default.
• W1808449080 startingPage "2419" @default.
• W1808449080 abstract "A contact-inhibited revertant of mink cells transformed by the Gardner-Arnstein strain of feline sarcoma virus was isolated by fluorescence-activated sorting of cells stained with the mitochondria-specific dye rhodamine 123. The revertant cell line exhibited a decrease in its proliferative rate and saturation density and a complete loss of its capacity for anchorage-independent growth, but it remained tumorigenic when inoculated into nude mice. The revertant cells retained a rescuable Gardner-Arnstein feline sarcoma provirus, expressed high levels of the v-fes oncogene product and its associated tyrosine kinase activity, manifested elevated levels of phosphotyrosine-containing cellular proteins similar to those observed in v-fes-transformed cells, and were refractory to retransformation by retroviruses containing the v-fes, v-fms, and v-ras oncogenes. Fusion of the revertant and parental cells generated somatic cell hybrids which formed colonies in semisolid medium, indicating that the block in transformation was recessive. These data together with the observation that the revertant phenotype is unstable in continuous culture suggest that the loss of transformation is due to the presence of limiting quantities of a gene product which functions downstream of the v-fes-coded kinase in the mitogenic pathway." @default.
• W1808449080 created "2016-06-24" @default.
• W1808449080 creator A5015143631 @default.
• W1808449080 creator A5017407539 @default.
• W1808449080 date "1988-06-01" @default.
• W1808449080 modified "2023-10-16" @default.
• W1808449080 title "A recessive cellular mutation in v-fes-transformed mink cells restores contact inhibition and anchorage-dependent growth." @default.
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• W1808449080 doi "https://doi.org/10.1128/mcb.8.6.2419" @default.
• W1808449080 hasPubMedCentralId "https://www.ncbi.nlm.nih.gov/pmc/articles/363440" @default.
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• W1808449080 hasPublicationYear "1988" @default.
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