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- W180917668 abstract "In 1850, Richard Quain reported in a series of 83 autopsied patients, some of whom in life were troubled by chest and arm pains, shortness of breath, syncopy and fits of coma, a peculiar patchy, pale dirty brown appearance to the surface of the heart on morbid anatomical inspection [1]. This change in color and consistency, which was accompanied by surface or interstitial fat, like ‘oil globules of milk bound in an albuminous envelope’, in a setting of friable muscle fibers ‘appeared to be connected with the causes on which the disease condition depend(ed) — such as obstruction of the coronary arteries’. Rokitansky and Virchow, both of whom thought (erroneously) that atherosclerosis and myocardial fibrosis were due to inflammation, felt this interlarding or fatty metamorphosis of cardiac muscle was an actual conversion of muscle fibrils to molecular fat [2, 3]. Virchow specifically advised that this formation of adipose tissue from connective tissue and parenchyma affected a derangement in motor power [4] or myomalacia cordes, a term that survived into the early twentieth century. From these rudimentary concepts evolved our modern understanding of myocardial ischemia, injury, and infarction and their interdependent relationships with fatty acids and altered lipid metabolism in heart muscle.KeywordsFree Fatty AcidFatty Acid Binding ProteinHeart CircAdenine Nucleotide TranslocaseFatty Acid AnalogueThese keywords were added by machine and not by the authors. This process is experimental and the keywords may be updated as the learning algorithm improves." @default.
- W180917668 created "2016-06-24" @default.
- W180917668 creator A5053009715 @default.
- W180917668 creator A5075626790 @default.
- W180917668 date "1990-01-01" @default.
- W180917668 modified "2023-09-25" @default.
- W180917668 title "Detrimental effects of fatty acids and their derivatives in ischemic and reperfused myocardium" @default.
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