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- W18199832 abstract "Intestinal functions are under the control of extrinsic afferent neurons, extrinsic efferent neurons of the sympathetic and parasympathetic autonomic nervous system and the intrinsic neurons of the enteric nervous system. Apart from the classical transmitters acetylcholine and noradrenaline, these neurons express a variety of neuropeptides including the tachykinins substance P (SP) and neurokinin A (NKA), calcitonin gene-related peptide (CGRP), vasoactive intestinal polypeptide (VIP) and neuropeptide Y [1]. Nerve activity leads to exocytotic release of the peptides which, by interacting with specific receptors on postjunctional neurons or effector cells, participate in the regulation of gastrointestinal motility, secretion, circulation and tissue homeostasis. After recognition of the physiological roles of peptidergic neurons and neuropeptides in the gut it is becoming increasingly evident that gastrointestinal inflammation and other disorders may be related to imbalanced function of certain neuropeptide systems. The conjecture that neuropeptides have a bearing on gastrointestinal disease has been most advanced for SP, NKA and CGRP, and it is with these peptides that the present article is concerned.KeywordsClostridium DifficileVasoactive Intestinal PolypeptideEnteric Nervous SystemPeptidergic NeuronClostridium Difficile ToxinThese keywords were added by machine and not by the authors. This process is experimental and the keywords may be updated as the learning algorithm improves." @default.
- W18199832 created "2016-06-24" @default.
- W18199832 creator A5005473061 @default.
- W18199832 date "1999-01-01" @default.
- W18199832 modified "2023-09-25" @default.
- W18199832 title "Neuropeptides and the gut: Tachykinins and calcitonin gene-related peptide in intestinal inflammation and pain" @default.
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- W18199832 doi "https://doi.org/10.1007/978-3-0348-8753-3_12" @default.
- W18199832 hasPublicationYear "1999" @default.
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