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- W18381874 abstract "Cerebral edema is defined as an increase in the volume of the brain due to an increase in brain water content. The water content of the brain in health is about 80% of wet weight for the gray matter, and 70% of wet weight for the white matter. The adverse effects of cerebral edema are not caused by the direct effects of the swelling itself, or on impaired neuronal transmission, or by disturbances in the chemical composition of brain tissue, or by deficits in oxygen delivery. Rather, the adverse effects appear to be indirect and mediated by cerebral ischemia, increased intracranial pressure, and brain shift with displacement. The manifestations of cerebral edema range from minimal to focal to generalized neurologic deficits. The dreaded complication of cerebral edema involves brain herniation. Various forms of brain herniation can occur, including cingulate herniation of the cingulate gyrus and the hemisphere beneath the falx to the contralateral side, uncal herniation of the medial portion of the temporal lobe through the tentorium with subsequent midbrain compression, tonsillar herniation of the cerebellum through the foramen magnum with brain stem compression, and transcalvarial herniation of any portion of the brain through craniotomy sites. Finally, weak diuretic agents have been tried in the treatment of cerebral edema. Thiazides, amiloride, and spironolactone, all lower cerebrospinal fluid production, but there are virtually no clinical studies to look at their utility in the treatment of cerebral edema." @default.
- W18381874 created "2016-06-24" @default.
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- W18381874 date "1997-01-01" @default.
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- W18381874 title "Diuretics and Cerebral Edema" @default.
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- W18381874 doi "https://doi.org/10.1016/b978-012635690-8/50032-3" @default.
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