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- W1838815905 abstract "ABSTRACT The endothelial cell interactions of homozygous null mutants of Candida albicans that were deficient in secreted aspartyl proteinase 1 (Sap1), Sap2, or Sap3 were investigated. Only Sap2 was found to contribute to the ability of C. albicans to damage endothelial cells and stimulate them to express E-selectin. None of the Saps studied appears to play a role in C. albicans adherence to endothelial cells." @default.
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- W1838815905 date "1998-06-01" @default.
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- W1838815905 title "Secreted Aspartyl Proteinases and Interactions of <i>Candida albicans</i> with Human Endothelial Cells" @default.
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- W1838815905 doi "https://doi.org/10.1128/iai.66.6.3003-3005.1998" @default.
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