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• W1840042037 abstract "Endotoxin (lipopolysaccharide [LPS]) released during gram-negative bacterial infection induces varieties of cytokines which directly and/or indirectly cause shock, disseminated intravascular coagulation, and death. We previously showed that lysozyme (LZM) was an LPS-binding protein and inhibited various immunomodulating activities of LPS. In this study, we examined the effect of LZM on the LPS-triggered septic shock model induced by carrageenan treatment and assessed by tumor necrosis factor production. The data presented in this report strongly suggest that LZM-LPS complex formation completely abrogates tumor necrosis factor production and the mortality caused by LPS and that LZM may be useful for the treatment of endotoxin shock." @default.
• W1840042037 created "2016-06-24" @default.
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• W1840042037 date "1994-04-01" @default.
• W1840042037 modified "2023-10-03" @default.
• W1840042037 title "Binding of lysozyme to lipopolysaccharide suppresses tumor necrosis factor production in vivo" @default.
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• W1840042037 doi "https://doi.org/10.1128/iai.62.4.1171-1175.1994" @default.
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