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• W1842247384 abstract "There have been different attempts with superantigen to modify experimental allergic encephalomyelitis (EAE), which is taken as an animal model for multiple sclerosis (MS). MS is not simply an inflammatory disease like EAE. It is well established that in MS chronic degeneration runs in parallel to outbreaks of inflammation. The portions of inflammation and degeneration may vary so that different types of disease course can be distinguished. Recent studies indicate that the degenerative process MS is not always secondary to but may also precede neuroinflammation. The chapter presents experiments which demonstrate that superantigen expressed in the brain or spinal cord is capable of inducing neuroinflammation as in MS. Horizontal sections of the cerebral hemispheres at the corpus callosum and at the level of the lateral ventricles were stained with hematoxylin/eosin. It is well known that relapsing-remitting MS patients with frequent relapses progress faster than patients with less frequent relapses. HLA associations identified in patients with a relapsing-remitting course of MS may be due to major histocompatibility complex (MHC)-specific binding properties of T-cell superantigens. Different so-called autoimmune diseases involve nerves or muscles. Pathogens like bacteria or viruses are involved in part of them. Although it is likely that true autoimmune phenomena following molecular mimicry are of pathogenetic relevance in some of these diseases, it seems possible that superantigens are involved in parallel or alone in a subgroup of patients with autoimmune neuritis or myositis." @default.
• W1842247384 created "2016-06-24" @default.
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• W1842247384 date "2014-04-08" @default.
• W1842247384 modified "2023-09-27" @default.
• W1842247384 title "Experimental Models of Superantigen-Mediated Neuropathology" @default.
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• W1842247384 doi "https://doi.org/10.1128/9781555815844.ch11" @default.
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