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- W1846601160 abstract "Patients with cirrhosis are prone to develop acute kidney injury (AKI) due to a number of causes, including bacterial infections with or without septic shock, hypovolemia, administration of nephrotoxic drugs, and intrinsic kidney diseases, among others. Most importantly, patients with advanced cirrhosis develop a distinctive cause of AKI, characterized by rapidly progressive glomerular filtration rate loss associated with marked disturbances in circulatory function in the absence of obvious pathologic abnormalities in the kidneys, known as hepatorenal syndrome (HRS). Decreased kidney function results from intense renal vasoconstriction secondary to the complex circulatory changes of cirrhosis with splanchnic vasodilatation and effective hypovolemia. Beyond activation of vasoactive systems, factors including impaired renal blood flow autoregulation and systemic inflammation may play a role in the development of HRS. Most patients improve with albumin and vasopressors; however, the prognosis of HRS remains very poor. Novel biomarkers may be helpful in distinguishing HRS from other causes of AKI in patients with cirrhosis." @default.
- W1846601160 created "2016-06-24" @default.
- W1846601160 creator A5019575404 @default.
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- W1846601160 creator A5033754624 @default.
- W1846601160 creator A5060242274 @default.
- W1846601160 creator A5070486020 @default.
- W1846601160 date "2016-02-01" @default.
- W1846601160 modified "2023-10-16" @default.
- W1846601160 title "Pathogenesis of Hepatorenal Syndrome: Implications for Therapy" @default.
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