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- W1849090336 abstract "Publisher Summary This chapter defines the vulnerability of the developing nervous system to organophosphate pesticides, in terms of cognitive functions, social and sex-related behavioral patterns, and body weight regulation. Impaired neurobehavioral development of children has been significantly linked in epidemiological studies with exposure to pesticides. Environmental epidemiological studies are essential for identifying toxic risks, but like any single scientific approach they have limitations. Determining the cause-and-effect relationship beyond significant association is a challenge. Significant behavioral alterations are reported after short-term, low-dose exposure to a variety of organophosphates during development. Because these effects were observed at doses that do not significantly inhibit acetylcholinesterase, other mechanisms in addition to inhibition of this important enzyme should be considered. The aim of toxicology is to be a predictive science, to prevent toxic damage. Animal model studies can work in concert with epidemiological research to resolve many of these issues. With regard to organophosphate (OP) pesticide-induced developmental neurobehavioral toxicity, laboratory animal model studies have clearly demonstrated that the developing nervous system is quite vulnerable to detrimental effects of OP pesticides, even if exposure was short-term and at doses that did not cause much inhibition of acetylcholinesterase." @default.
- W1849090336 created "2016-06-24" @default.
- W1849090336 creator A5004763783 @default.
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- W1849090336 date "2010-01-01" @default.
- W1849090336 modified "2023-09-26" @default.
- W1849090336 title "Lasting Behavioral Consequences of Organophosphate Pesticide Exposure During Development" @default.
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- W1849090336 doi "https://doi.org/10.1016/b978-0-12-374367-1.00033-1" @default.
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