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- W184952641 abstract "Pseudomonas aeruginosa infections may develop in immunocompromised patients in almost every tissue/organ, including: skin, eyes, ears, nasopharynx, bronchi and lungs, heart, kidneys and urinary tract, intestinal tract, liver, spleen, bones, brain and blood (bacteremia). They are the major cause of death in cystic fibrosis patients and endanger the life of patients suffering from extensive burns, chronic diseases and immunosuppressive ailments and treatments (e.g., for cancer repression or tissue transplantations). Their endurance is dependent on the tissue/host background. They are generally secondary to either immunodeficiency and primary infections or to metabolic abnormalities, which alter cell surface composition, exposing galactose- or N-acetylgalactosamine-bearing glycolipids. In primary infections preceding them, such as influenza, there is either removal of sialic acids or glycopeptides by the infecting factor sialidases or proteases. In metabolic abnormalities such as cystic fibrosis, the reason for the reduced glycolipid sialylation may be either sialylation deficiency (Imundo et al., 1995) or injurious sialo-/proteolysis caused by salivary or leukocytic enzymes." @default.
- W184952641 created "2016-06-24" @default.
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- W184952641 date "1996-01-01" @default.
- W184952641 modified "2023-09-26" @default.
- W184952641 title "Towards Anti- Pseudomonas Aeruginosa Adhesion Therapy" @default.
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- W184952641 doi "https://doi.org/10.1007/978-1-4613-0415-9_5" @default.
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