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- W185372220 abstract "Hyponatremia is the most common electrolyte disorder observed in hospitalized patients, with an incidence of about 1.5% [1]. In virtually all clinical situations characterized by hyponatremia, renal excretion of water is impaired, and this is most commonly due to elevations in the plasma level of antidiuretic hormone (ADH). The most common exceptions to this are two situations: a) acute water intoxication; b) idiosyncratic reaction to either aldersterone-inhibiting or thiazide diuretics — where plasma ADH is normal to low. The major stimuli for physiologic increases in plasma ADH are abnormalities where fluid volume is low, or tonicity (osmolality) is elevated. There are many clinical situations where, in the absence of a “logical” physiologic reason, such as decreased fluid volume or increased tonicity, plasma ADH is abnormally elevated [2]. In such situations, the ADH is said to be elevated “inappropriately”, as in the syndrome of inappropriately elevated plasma levels of antidiuretic hormone (SIADH). Possible stimuli for the inappropriate increase of ADH include multiple non-osmotic stimuli for increased ADH secretion (pain, fear, bleeding, anesthesia, narcotics, barbiturates) and certain neoplasms which secrete either vasopressin or a vasopressin-like Polypeptide [3]. Hyponatremia is commonly seen in association with many different systemic disease states, the most common of which are hepatic cirrhosis, congestive heart failure and nephrotic syndrome." @default.
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- W185372220 date "1989-01-01" @default.
- W185372220 modified "2023-09-27" @default.
- W185372220 title "Treatment of Symptomatic Hyponatremia and Permanent Brain Lesions" @default.
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- W185372220 doi "https://doi.org/10.1007/978-3-642-83737-1_34" @default.
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