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• W1867394908 abstract "In this review, we consider the role of endocannabinoids and cannabinoid‐1 (CB 1 ) cannabinoid receptors in metabolic regulation and as mediators of the thrifty phenotype that underlies the metabolic syndrome. We survey the actions of endocannabinoids on food intake and body weight, as well as on the metabolic complications of visceral obesity, including fatty liver, insulin resistance and dyslipidemias. Special emphasis is placed on weighing the relative importance of CB 1 receptors located in peripheral tissues versus the central nervous system in mediating the metabolic effects of endocannabinoids. Finally, we review recent observations that indicate that peripherally restricted CB 1 receptor antagonists retain efficacy in reducing weight and improving metabolic abnormalities in mouse models of obesity without causing behavioural effects predictive of neuropsychiatric side effects in humans. LINKED ARTICLES This article is part of a themed issue on Cannabinoids in Biology and Medicine. To view the other articles in this issue visit http://dx.doi.org/10.1111/bph.2011.163.issue‐7" @default.
• W1867394908 created "2016-06-24" @default.
• W1867394908 creator A5026132845 @default.
• W1867394908 creator A5064634234 @default.
• W1867394908 date "2011-07-12" @default.
• W1867394908 modified "2023-09-23" @default.
• W1867394908 title "The case for peripheral CB₁receptor blockade in the treatment of visceral obesity and its cardiometabolic complications" @default.
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• W1867394908 doi "https://doi.org/10.1111/j.1476-5381.2011.01352.x" @default.
• W1867394908 hasPubMedCentralId "https://www.ncbi.nlm.nih.gov/pmc/articles/3165952" @default.
• W1867394908 hasPubMedId "https://pubmed.ncbi.nlm.nih.gov/21434882" @default.
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