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• W1870005427 abstract "Angiogenesis induced by proangiogenic molecules such as vascular endothelial growth factor (VEGF) is a key process in the progression and metastasis of gastric cancer. In this study, we investigated the role of VEGF-C/VEGF receptor 3 (VEGFR3) axis in cell proliferation and migration/invasion of human gastric cancer cells (hGCCs). VEGF-C did not enhance cell proliferation but increased cell migration/invasion by approximately ∼50% in hGCCs (AGS and SNU-484). MAZ51, a VEGFR3 inhibitor, reduced the VEGF-C-induced increase in migration/invasion by ∼30% (p < 0.05). These results suggest that VEGFR3 could be a therapeutic target for reducing the metastasis of gastric cancer cells." @default.
• W1870005427 created "2016-06-24" @default.
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• W1870005427 date "2015-06-26" @default.
• W1870005427 modified "2023-10-18" @default.
• W1870005427 title "Inhibition of Vascular Endothelial Growth Factor Receptor 3 Reduces Migration of Gastric Cancer Cells" @default.
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• W1870005427 doi "https://doi.org/10.3109/07357907.2015.1047509" @default.
• W1870005427 hasPubMedId "https://pubmed.ncbi.nlm.nih.gov/26115478" @default.
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