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- W1876201922 endingPage "426" @default.
- W1876201922 startingPage "416" @default.
- W1876201922 abstract "In the nervous system, neurons contact each other to form neuronal circuits and drive behavior, relying heavily on synaptic connections. The proper development and growth of synapses allows functional transmission of electrical information between neurons or between neurons and muscle fibers. Defects in synapse-formation or development lead to many diseases. Autophagy, a major determinant of protein turnover, is an essential process that takes place in developing synapses. During the induction of autophagy, proteins and cytoplasmic components are encapsulated in autophagosomes, which fuse with lysosomes to form autolysosomes. The cargoes are subsequently degraded and recycled. However, aberrant autophagic activity may lead to synaptic dysfunction, which is a common pathological characteristic in several disorders. Here, we review the current understanding of autophagy in regulating synaptic development and function. In addition, autophagy-related synaptic dysfunction in human diseases is also summarized." @default.
- W1876201922 created "2016-06-24" @default.
- W1876201922 creator A5025201072 @default.
- W1876201922 creator A5051970233 @default.
- W1876201922 creator A5055118791 @default.
- W1876201922 creator A5072690470 @default.
- W1876201922 date "2015-07-02" @default.
- W1876201922 modified "2023-10-16" @default.
- W1876201922 title "Autophagy in synaptic development, function, and pathology" @default.
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