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- W1877736066 abstract "Extracellular adenosine triphosphate (ATP) has potent systemic vasodilator and endothelial-dependent relaxant effects on precontracted vessels. Pulmonary uptake and metabolism of ATP have been described, but experimental effects on pulmonary vessels remain controversial in animals. The effects of an intravenously administered infusion of ATP on pulmonary hemodynamic and gasometric data were assessed in 18 patients with stable chronic obstructive pulmonary disease (COPD). Low doses of ATP (successive rates, 1 and 2 [µmol/kg body weight, each for 20 min) were infused in pulmonary hypertensive (H; n = 6) and nonhypertensive (N; n = 6) patients. They were compared with a control group (C; n = 6) that received only solvent, using ANOVA. During ATP infusion, a significant pulmonary vasodilation was demonstrated as simultaneous decreases reached, respectively, −14.2% (Group H; p < 0.005) and −13.8% (Group N; p < 0.001) for mean pulmonary artery pressure (), and −31.7% (H; p < 0.05) and −20.7% (N; p < 0.01) for p..." @default.
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- W1877736066 date "2015-05-14" @default.
- W1877736066 modified "2023-09-23" @default.
- W1877736066 title "Effects of Adenosine Triphosphate on Pulmonary Circulation in Chronic Obstructive Pulmonary Disease" @default.
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