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- W187898767 endingPage "282" @default.
- W187898767 startingPage "265" @default.
- W187898767 abstract "The p53 tumor suppressor gene and gene product are among the most diverse and complex been shown to have a direct correlation with cancer development and have been shown to occur in nearly 50% of all cancers. p53 mutations are particularly common in skin cancers and UV irradiation has been shown to be a primary cause of specific 'signature' mutations that can result in oncogenic transformation. There are certain 'hot-spots' in the p53 gene where mutations are commonly found that result in a mutated dipyrimidine site. This review discusses the role of p53 from normal function and its dysfunction in precancerous lesions, nonmelanoma and melanoma skin cancers. Additionally, molecules that associate with p53 and alter its function to produce neoplastic conditions are also explored in this chapter." @default.
- W187898767 created "2016-06-24" @default.
- W187898767 creator A5003800717 @default.
- W187898767 creator A5013793536 @default.
- W187898767 creator A5019080361 @default.
- W187898767 date "2009-01-07" @default.
- W187898767 modified "2023-10-01" @default.
- W187898767 title "p53 Protein and Pathogenesis of Melanoma and Nonmelanoma Skin Cancer" @default.
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