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- W1879521737 abstract "This editorial refers to ‘A novel human R25C-phospholamban mutation is associated with super-inhibition of calcium cycling and ventricular arrhythmia’ by G.-S. Liu et al. , pp. 164–174. Genetically caused dilated cardiomyopathy (DCM) is typically diagnosed between 40 and 60 years and patients often present with heart failure or advanced arrhythmia, which may possibly lead to stroke or sudden cardiac death.1 While potentially lethal arrhythmias are commonly found in end-stage heart failure independent of its aetiology, there is growing evidence that in certain cases of DCM, arrhythmia and sudden cardiac death are present already in early stages and may precede the development of overt heart failure. So far, this subset of cases has been linked to mutations in LMNA , coding for lamin A/C, and SCN5A , coding for the cardiac fast Na+ channel α-subunit NaV1.5.1 In the recent years, also a mutation in PLN , coding for phospholamban (PLB), R14del, has been connected to this subset of DCM.2–4PLB is the main regulator of SERCA …" @default.
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- W1879521737 date "2015-05-26" @default.
- W1879521737 modified "2023-09-26" @default.
- W1879521737 title "Linking superinhibitory PLN mutations to CaMKII activation: a new arrhythmogenic mechanism in genetic DCM?" @default.
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- W1879521737 doi "https://doi.org/10.1093/cvr/cvv163" @default.
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