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- W189289905 abstract "Rheumatoid Arthritis (RA) is a systemic disease of unknown etiology, with the main manifestation being chronic inflammation in multiple joints. In many patients the arthritis is characterized by exacerbations and remissions. Current theories of the pathogenesis of the chronic arthritis include a sustained immune reaction directed at unknown exogenous agents that localize to the joints or articular autoantigens such as cartilage matrix molecules. In addition, chronicity might be linked to a deranged “tumor like” behaviour of the synovial tissue cells of RA patients, including macrophages and synovial fibroblasts. These cells show an aggressive phenotype and prolonged growth in culture, without an apparent need for further stimuli. It might be argued that the underlying trigger is co-isolated with the cells, still being present in the culture and hence creating a seemingly autonomous activation. In that sense, viruses may be likely candidates." @default.
- W189289905 created "2016-06-24" @default.
- W189289905 creator A5045199963 @default.
- W189289905 date "1998-01-01" @default.
- W189289905 modified "2023-09-27" @default.
- W189289905 title "Role of T cells in arthritis: Lessons from animal models" @default.
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- W189289905 doi "https://doi.org/10.1007/978-3-0348-8823-3_4" @default.
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