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- W189426722 abstract "Although gastroparesis represents a significant health care burden, it lacks effective and curative therapy. Studies in animal models have identified degenerative changes in the gastric tunica muscularis, such as loss of interstitial cells of Cajal and nitrergic neurotransmission as major pathogenetic factors in this disorder. Recently, the same abnormalities were detected in relatively large cohorts of patients with refractory gastroparesis. This chapter reviews how these and other cellular changes can lead to the development of gastroparesis. It is proposed that gastroparesis develops from an interaction between chronic cell depletion and acute functional challenges. Future therapeutic approaches should focus on restoring tissue integrity by preventing or countering cell degeneration or by replacing the missing cells." @default.
- W189426722 created "2016-06-24" @default.
- W189426722 creator A5031123252 @default.
- W189426722 date "2011-08-24" @default.
- W189426722 modified "2023-10-16" @default.
- W189426722 title "Cellular Pathogenesis of Gastroparesis" @default.
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- W189426722 doi "https://doi.org/10.1007/978-1-60761-552-1_5" @default.
- W189426722 hasPublicationYear "2011" @default.
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