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• W1901123133 startingPage "147" @default.
• W1901123133 abstract "Summary: Large numbers of environmental antigens, including commensal bacteria and food‐derived antigens, constitutively interact with the epithelial layer of the gastrointestinal (GI) tract. Commensal bacteria peacefully cohabit with the host GI tract and exert multiple beneficial or destructive effects on their host. Intestinal epithelial cells (IECs) constitute the first physical and immunological protective wall against invasive pathogens and a cohabitation niche for commensal bacteria. As the physiological homeostasis of IECs is maintained by multiple biological processes such as apoptosis, autophagy, and the handling of endoplasmic reticulum stress, the aberrant kinetics of these biological events, which have genetic and environmental causes, leads to the development of host intestinal pathogenesis such as inflammatory bowel disease. In addition, IECs recognize and interact with commensal bacteria and give instructions to mucosal immune cells to initiate an immunological balance between active and quiescent conditions, eventually establishing intestinal homeostasis. The mucosal immune system regulates the homeostasis of gut microbiota by producing immunological molecules such as secretory immunoglobulin A, the production of which is mediated by IECs. IECs therefore play a central role in the creation and maintenance of a physiologically and immunologically stable intestinal environment." @default.
• W1901123133 created "2016-06-24" @default.
• W1901123133 creator A5001003639 @default.
• W1901123133 creator A5087178931 @default.
• W1901123133 date "2011-12-15" @default.
• W1901123133 modified "2023-10-07" @default.
• W1901123133 title "Epithelial barrier: an interface for the cross-communication between gut flora and immune system" @default.
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