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- W19037578 abstract "This chapter discusses the biochemical methods to study physiological functions of thyrocalcitonin. Parathyroid hormone is virtually essential for life. If the source of the endogenous hormone, the parathyroid glands, is removed, the plasma calcium falls to a dangerously low level and if appropriate therapy is not administered the animal may die. It is apparent that parathyroid hormone must be secreted essentially continuously in order to protect against hypocalcemia. In contrast to parathyroidectomy, thyroidectomy does not result in any acute change in the blood calcium level in rats and other animals maintained in the fasting state. The hypocalcemia resulting from parathyroidectomy is not matched by hypercalcemia after thyroidectomy. The lower the plasma calcium, the less calcium is lost in the urine. Rats with intact thyroid glands excrete very little calcium, whereas thyroidectomized rats excrete a relatively tremendous amount—20 times as much. The most likely threat of hypercalcemia in healthy animals occurs during absorption of calcium from the diet. The normal thyroid gland responds quickly to the presence of calcium in the gastrointestinal tract by increasing the secretion of thyrocalcitonin that in turn prevents or restricts hypercalcemia by its action on bone." @default.
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- W19037578 date "1972-01-01" @default.
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- W19037578 title "Physiological Function of Thyrocalcitonin" @default.
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