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- W1920185087 abstract "We appreciate Professor Rumboldt’s interest in our article and his caring comments (1). As he said, most published articles propose mitochondrial damage as the mechanism of phosphine toxicity; in fact, this hypothesis has been repeated in almost every medical article on aluminium phosphide (AlP) poisoning since the 1990s (24). To our knowledge, it has not been backed up by any studies in humans. Moreover, in vitro studies indicate incomplete inhibition of the mitochondrial function (5). As we know, the main problem with severe AlP poisoning is refractory hypotension, which is almost always associated with severe refractory metabolic acidosis and fatal prognosis (6). There is a general consensus that the only possible management is supportive. However, despite advanced intensive care strategies, mortality rates vary between 24 and 66 % within the first few days (7, 8). This urged us to ask ourselves the following:" @default.
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- W1920185087 date "2015-09-01" @default.
- W1920185087 modified "2023-09-26" @default.
- W1920185087 title "Response to Professor Rumboldt’s reaction to our letter on hydroxyethyl starch use in managing aluminium phosphide poisoning" @default.
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- W1920185087 doi "https://doi.org/10.1515/aiht-2015-66-2649" @default.
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