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- W1922024307 abstract "Bacteria proliferate by repetitive cycles of cellular growth and division. The progression into the cell cycle is admitted to be under the control of cell size. However, the molecular basis of this regulation is still unclear. Here I will discuss which mechanisms could allow coupling growth and division by sensing size and transmitting this information to the division machinery. Size sensors could act at different stages of the cell cycle. During septum formation, mechanisms controlling the formation of the Z ring, such as MinCD inhibition or Nucleoid Occlusion (NO) could participate in the size-dependence of the division process. In addition or alternatively, the coupling of growth and division may occur indirectly through the control of DNA replication initiation. The relative importance of these different size-sensing mechanisms could depend on the environmental and genetic context. The recent demonstration of an incremental strategy of size control in bacteria, suggests that DnaA-dependent control of replication initiation could be the major size control mechanism limiting cell size variation." @default.
- W1922024307 created "2016-06-24" @default.
- W1922024307 creator A5019347263 @default.
- W1922024307 date "2015-05-29" @default.
- W1922024307 modified "2023-10-17" @default.
- W1922024307 title "Size sensors in bacteria, cell cycle control, and size control" @default.
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- W1922024307 doi "https://doi.org/10.3389/fmicb.2015.00515" @default.
- W1922024307 hasPubMedCentralId "https://www.ncbi.nlm.nih.gov/pmc/articles/4448035" @default.
- W1922024307 hasPubMedId "https://pubmed.ncbi.nlm.nih.gov/26074903" @default.
- W1922024307 hasPublicationYear "2015" @default.
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