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- W1922443769 abstract "Rabbits immunized against prostaglandins develop antibodies to prostaglandins and peptic ulcer disease (duodenal and gastric ulcers). We have evaluated the hypothesis that idiopathic gastric or duodenal ulcer disease in humans may be associated with the spontaneous occurrence of serum antibodies directed against endogenous prostaglandins. We found that serum from 45 ulcer patients (34 duodenal, 11 gastric) had a low degree of binding of radiolabeled prostaglandin E2, prostaglandin F2 alpha, or 6-keto prostaglandin F1 alpha. The extent to which prostaglandins were bound to serum of ulcer patients was not statistically different from prostaglandin binding to serum from 25 normal subjects. Therefore, we conclude that spontaneous occurrence of circulating antibodies against endogenous prostaglandins is an unlikely cause of gastroduodenal ulceration in humans." @default.
- W1922443769 created "2016-06-24" @default.
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- W1922443769 date "1986-07-01" @default.
- W1922443769 modified "2023-09-23" @default.
- W1922443769 title "Lack of specific binding of prostaglandin E2, prostaglandin F2α, and 6-Keto Prostaglandin F1α to serum in patients with peptic ulcer disease and in healthy subjects" @default.
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- W1922443769 doi "https://doi.org/10.1016/0016-5085(86)90440-3" @default.
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