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• W1922613042 abstract "RIIIS/J mice lack an autosomal dominant gene(s) that influences the magnitude of the antibody response to several polysaccharide antigens of bacterial origin. Low responsiveness is demonstrable whether polysaccharide is administered as a T-helper-cell-independent or -dependent antigen conjugated to an immunogenic carrier; however, RIIIS/J mice make good anti-hapten antibody responses to haptenated polysaccharides. The low antibody responses of RIIIS/J mice to type III pneumococcal polysaccharide do not appear to be the results of an imbalance in the activity of regulatory T lymphocytes. Compared with other strains of mice, RIIIS/J mice elicit low antibody responses to lipopolysaccharide (LPS). They do not develop a cyclic primary or secondary antibody response to Escherichia coli O113 LPS; the latter is not due to a lack of mitogenic response to E. coli O113 LPS. They also produce auto-anti-idiotypic antibody after being immunized with trinitrophenyl-Ficoll." @default.
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• W1922613042 date "1990-05-01" @default.
• W1922613042 modified "2023-09-27" @default.
• W1922613042 title "Characterization of the immunodeficiency of RIIIS/J mice: immune response to polysaccharide antigens" @default.
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• W1922613042 doi "https://doi.org/10.1128/iai.58.5.1261-1268.1990" @default.
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