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• W1929126069 endingPage "621" @default.
• W1929126069 startingPage "609" @default.
• W1929126069 abstract "Damage to the central nervous system, as in the case of spinal cord injury (SCI), results in disrupted supraspinal sympathetic influence and subsequent cardiovascular control impairments. Consequently, people with SCI suffer from disordered basal hemodynamics and devastating fluctuations in blood pressure, as in the case of autonomic dysreflexia (AD), which likely contribute to this population's leading cause of mortality: cardiovascular disease. The development of AD is related, at least in part, to neuroanatomical changes that include disrupted descending supraspinal sympathetic control, changes in propriospinal circuitry, and inappropriate afferent sprouting in the dorsal horn. These anatomical mechanisms may thus be targeted by neural regenerative and protective therapies to improve cardiovascular control and reduce AD. Here, we discuss the relationship between abnormal cardiovascular control and its underlying neuroanatomy. We then review current studies investigating biochemical strategies to reduce the severity of AD through: 1) reducing aberrant calcitonin gene-related peptide immunoreactive afferent sprouting; 2) inhibiting inflammatory processes; and 3) re-establishing descending supraspinal sympathetic control. Finally, we discuss why additional biochemical agents and combinational approaches may be needed to completely ameliorate this condition." @default.
• W1929126069 created "2016-06-24" @default.
• W1929126069 creator A5004672062 @default.
• W1929126069 creator A5023450351 @default.
• W1929126069 creator A5036626766 @default.
• W1929126069 date "2015-05-01" @default.
• W1929126069 modified "2023-09-25" @default.
• W1929126069 title "Neuroprotection, Plasticity Manipulation, and Regenerative Strategies to Improve Cardiovascular Function following Spinal Cord Injury" @default.
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• W1929126069 doi "https://doi.org/10.1089/neu.2014.3743" @default.
• W1929126069 hasPubMedId "https://pubmed.ncbi.nlm.nih.gov/25582334" @default.
• W1929126069 hasPublicationYear "2015" @default.
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