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- W1937223434 endingPage "kev337" @default.
- W1937223434 startingPage "kev337" @default.
- W1937223434 abstract "Autophagy appears to play a dual role in eukaryotic cells. It manifests cytoprotective effects through the regulation of catabolic processes and the clearance of pathogens; however, a correlation between autophagy and the pathogenesis of autoimmune/autoinflammatory conditions has recently been described. Autophagy has emerged as a mediator in the pathogenesis of RA. Autophagy may regulate apoptosis resistance and hyperplasia in synovial fibroblasts, promote osteoclastogenesis and stimulate osteoclast-mediated bone resorption through the delivery of citrullinated peptides to MHC compartments, which results in the activation of the innate and adaptive immune response, thereby resulting in RA. Given the likely importance of autophagy in the pathogenesis of RA, here we reviewed the detailed mechanisms concerning the pathogenicity of autophagy and autophagy proteins in RA." @default.
- W1937223434 created "2016-06-24" @default.
- W1937223434 creator A5032024736 @default.
- W1937223434 creator A5045525711 @default.
- W1937223434 date "2015-09-05" @default.
- W1937223434 modified "2023-09-23" @default.
- W1937223434 title "Recent insights into the role of autophagy in the pathogenesis of rheumatoid arthritis" @default.
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- W1937223434 doi "https://doi.org/10.1093/rheumatology/kev337" @default.
- W1937223434 hasPubMedId "https://pubmed.ncbi.nlm.nih.gov/26342228" @default.
- W1937223434 hasPublicationYear "2015" @default.
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