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- W194460670 abstract "Catecholamines serve as neurotransmitters and adrenal hormones. The activity of pineal N-acetyltransferase is transsynaptically regulated by norepinephrine. N-acetyltransferase activity controls large daily changes in the amount of serotonin, and the amount and rate of production of N-acetylserotonin and melatonin in the pineal gland. This chapter describes the studies suggesting that sympathetic nerve endings in the pineal gland might play a protective role in stress. They act by removing circulating catecholamines from extracellular spaces, thus preventing an increase in N-acetyltransferase activity during stress. The mechanism through which catecholamines are taken up by nerve endings has been well characterized. It functions physiologically to terminate transsynaptic transmission by removing active neurotransmitter from extracellular spaces. This mechanism plays a protective role in stress. Circulating catecholamines, which are released because of stress, pass through blood vessels into the extracellular space of the pineal gland." @default.
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- W194460670 date "1976-01-01" @default.
- W194460670 modified "2023-09-26" @default.
- W194460670 title "A PROTECTIVE ROLE OF NERVE ENDINGS IN THE STRESS-STIMULATED INCREASE IN PINEAL N-ACETYLTRANSFERASE ACTIVITY" @default.
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- W194460670 doi "https://doi.org/10.1016/b978-0-08-020588-5.50021-7" @default.
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